[TheSutterDynasty]

Originally Posted by blankall

Excuse me? I have a degree in cell biology. I understand the terms.

Apparently not. I said:

Originally Posted by me

Saturated fats are never good for you. They raise LDL substantially. So no, bacon and fatty steaks are still terrible for you.

You said:

Originally Posted by blankall

The vast majority of cholesterol in your body is produced by the body itself. So while eating cholesterol may result in a very minor and temporary spike in cholesterol levels, it won't affect your overall levels over a period of time.

I said eating saturated fats increases LDL, but you said eating dietary cholesterol doesn't affect your overall serum lipids.

So no, apparently you don't understand the terms.

You also said:

Originally Posted by blankall

A lot of studies in the past showed there was a link between LDL consumption and heart disease, however, these studies did not take into account the affect of obesity and foods high in cholesterol are usually associated with high calorie diets.

LDL consumption? Low-density lipoproteins are the lipid transporting particles in our bodies. Maybe you should read up on them: http://en.wikipedia.org/wiki/LDL

And again you're talking about foods high in cholesterol. Saturated fat is not cholesterol.

This is cholesterol:

This is (one type of) saturated fat:

Hopefully your degree in cell biology can help you comprehend the differences.

Originally Posted by blankall

Nobody know the mechanism that causes heart disease. It's linked to obesity and more specifically visceral fat, but nobody knows the exact mechanims.

Hahaha are you kidding? I took an entire course based on the mechanisms of heart disease. Or is that "controversial" now too?

Here's a quick run down for you:

1. There is a disruption in the endothelial layer (this is why hypertension is a risk factor for heart disease)

2. LDL/VLDL/chylomicrons (TGs) enter and accumulate within the arterial wall and are oxidized and modified

3. This modified LDL (and VLDL and TGs) stimulates MCP-1, which attracts monocytes into the subendothelial space (inflammation is occurring)

4. LDL et al promote the differentiation of the monocytes into macrophages, which engulf the modified LDL et al and transform them into foam cells

5. These foam cells are the "plaque" that accumulates in the artery, causing atherosclerosis; heart disease.

6. HDL is good cholesterol because it promotes the efflux of cholesterol from the subendothelial space, and inhibits the oxidation of LDL et al.

Put that cell biology degree to work.

More to come..